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Sci Rep 10, Download citation. Received : 02 January Accepted : 15 June There was, however, a significantly greater tendency for those with nontransmural infarct to have evidence of prior infarction at autopsy 27 of 35 versus 19 of 35, p less than 0. Acute coronary thrombi in the distribution of the infarct were significantly more common among transmural myocardial infarcts 32 of 35 versus 18 of 35, p less than 0.
Morphologically, the nontransmural infarcts showed mural involvement ranging from 20 to 90 percent of the left ventricle, and histologically showed more contraction band i.
Fatal nontransmural and transmural infarcts have major clinical and pathologic similarities, but differences in number of prior infarcts, type of necrosis, and occurrence of coronary thrombi suggest differing pathophysiology. Hours to days later during the evolving phase, pathological Q waves appear, the elevated ST segments return towards baseline, and the T waves become inverted. Significant Q wave usually persists even after recovery. Localization of myocardial infarction By way of their position, the 12 ECG leads can be used to distinguish myocardial infarction occurring in different regions of the heart.
These uncharacteristic signs make the diagnosis of posterior MI difficult without heightened vigilance. Suffice it to say, pure posterior wall infarctions are rare.
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